Aquaculture 2022

February 28 - March 4, 2022

San Diego, California

ADVANCES IN UNDERSTANDING SOYBEAN MEAL INDUCED DISTAL ENTERITIS IN SALMONIDS

 

Madison S. Powell* ,  Brian Small,  T. Gibson Gaylord,  Wendy M. Sealey, Kenneth E. Overturf and Thomas Welker

 

Aquaculture Research Institute, University of Idaho, Hagerman Fish Culture Experiment Station,

 3059F National Fish Hatchery Road, Hagerman, ID 83332 USA

*mpowell@uidaho.edu

 



 Despite the promising amino-acid profile of soybean meal , inflammatory reactions arising from its inclusion in aquafeeds becomes a limiting factor to increased utilization of this alternative plant-based ingredient in salmonids . Numerous anti-nutritional factors  commonly present in plant-based ingredients have been recognized and studied as potential c auses for the inflammatory effects related to  soy-induced  distal enteritis. Efforts to address poor performance using plant-based diets include selection in a strain of rainbow trout (Oncorhynchus mykiss) on a 100% plant-meal based feed containing 19.6% SBM and 25.6% soy protein concentrate. In addition to improved growth performance, this selected strain has shown  concomitant resistance to the development of distal enteritis and evidence of increased oral tolerance to SBM. Several  recent  collaborative studies between the Hagerman Fish Culture Experiment Station and Bozeman Fish Technology Center highlight  work that has been done to characterize soybean meal induced distal enteritis and develop a clinical model for early detection, progression, and amelioration of adverse effects.

Resistance to distal enteritis observed in  the Hagerman  selected-strain rainbow trout provide an excellent contrast  and model  for further characterization of acute mucosal inflammatory responses involving IL-17 paralogs, other interleukins, growth factors, calcium-binding proteins and  other immunogenic markers . Additional studies have also characterized induction of T helper17 cells and the repression of T regulatory cells involved in oral tolerance as well as changes to gut histology and microbiota.