Aquaculture Africa 2023

November 13 - 16, 2023

Lusaka, Zambia

COPING MECHANISMS OF ADULT NILE TILAPIA Oreochromis niloticus EXPOSED TO HIGH ENVIRONMENTAL AMMONIA

Stephen Gyamfi*, Regina E. Edziyie, Kwasi A. Obirikorang, Daniel Adjei-Boateng   and Peter V. Skov

 Department of Fisheries and Watershed Management,

Kwame Nkrumah University of Science and Technology, Kumasi, Ghana

stephengyamfi40@gmail.com



Fish exhibit different behavioural and physiological coping mechanisms to unionized ammonia (UIA). Some species detoxify ammonia to less toxic glutamine or urea, or  they  excrete  it  directly into the aqueous environment via  the gills. The strategies adopted by Nile tilapia (Oreochromis niloticus) to UIA exposure, however remain vague.  To investigate the coping mechanisms of O. niloticus to  environmental ammonia, adult individuals were exposed to three levels of UIA, 0 (Control), 7 and 61 µM for 24,72 and168 h and assessing histological changes in the gills  and alterations in haematological parameters. 

ILCM

Nile tilapia with an average mass  of 281.1±34.0g  were exposed to 2 different levels of UIA for up to 168h. One group was exposed to 7µM NH3, and one was exposed to 61µM NH3 , and compared against a control  group.  Experimental fish were placed in individual  PVC tubes in  an  experimental tank . For the control group, eight fish were used .  For exposure to 7 and 61µM NH3 groups of fish (n=8)  were exposed for 24, 72  or  168h respectively.  At the end of the exposure period,  fish were euthan ized and blood, gill, liver and white  skeletal muscle was sampled from each fish . 

Blood pH showed a significant  (p<0.05) downregulation for each ammonia level exposure. R ed blood cells on the other hand increased from the control fish group after exposing Nile tilapia to 61µM for 168h. Nile tilapia responded to ammonia toxicity by significantly increasing plasma Cl- levels.  Exposure to  61µM  UIA resulted to a significant  increase in plasma NH3 and Na+. Liver and muscle urea decreased significantly from the control fish group with a significant increase in excreted urea only after 24h of exposing fish to 61 µM-UIA. T he groups exposed to 7µM showed a  tendency towards a  reduction in  interlamellar cell mass (ILCM)  for  all exposure durations ,  although  the reduction was not significant. I n response to 61µM UIA, ILCM  decreased significantly after 24 and 72h (Fig 1). A t higher UIA levels, gill remodelling occurs in Nile tilapia in response to ammonia exposure. It is also known from this experiment that Nile tilapia detoxify ammonia to urea but are mostly excreted in the aqueous environment.